Search results for "MESH : Phenotype"

showing 2 items of 2 documents

Arabidopsis thaliana nicotianamine synthase 4 is required for proper response to iron deficiency and to cadmium exposure.

2013

International audience; The nicotianamine synthase (NAS) enzymes catalyze the formation of nicotianamine (NA), a non-proteinogenic amino acid involved in iron homeostasis. We undertook the functional characterization of AtNAS4, the fourth member of the Arabidopsis thaliana NAS gene family. A mutant carrying a T-DNA insertion in AtNAS4 (atnas4), as well as lines overexpressing AtNAS4 both in the atnas4 and the wild-type genetic backgrounds, were used to decipher the role of AtNAS4 in NA synthesis, iron homeostasis and the plant response to iron deficiency or cadmium supply. We showed that AtNAS4 is an important source for NA. Whereas atnas4 had normal growth in iron-sufficient medium, it dis…

0106 biological sciences[ SDV.BV ] Life Sciences [q-bio]/Vegetal BiologyMESH : Azetidinecarboxylic AcidFMN ReductaseArabidopsis thalianaMutantArabidopsisGene ExpressionPlant Science01 natural sciencesMESH : Cation Transport ProteinsMESH : IronMESH : Arabidopsis ProteinsNicotianamine synthaseMESH : Plants Genetically Modifiedchemistry.chemical_compoundMESH : ArabidopsisGene Expression Regulation PlantGene expressionMESH: Genes PlantArabidopsis thalianaMESH : DNA BacterialHomeostasisMESH: ArabidopsisNicotianamineMESH: Stress PhysiologicalCation Transport ProteinsMESH : Adaptation PhysiologicalMESH : Cadmium2. Zero hungerchemistry.chemical_classification0303 health sciencesCadmiumMESH: IronbiologyGeneral MedicineIron DeficienciesPlants Genetically ModifiedAdaptation PhysiologicalMESH: Azetidinecarboxylic AcidMESH : PhenotypePhenotypeBiochemistryMESH: HomeostasisMESH : HomeostasisMESH : MutationAzetidinecarboxylic AcidCadmiumDNA BacterialMESH: Gene ExpressionMESH: MutationIronMESH: Cadmiumchemistry.chemical_elementMESH: FerritinsMESH: Arabidopsis ProteinsMESH: Alkyl and Aryl TransferasesGenes PlantMESH: PhenotypeNicotianamine synthase03 medical and health sciencesMESH: Cation Transport ProteinsStress PhysiologicalIron homeostasisGenetics[SDV.BV]Life Sciences [q-bio]/Vegetal BiologyIron deficiency (plant disorder)MESH: Gene Expression Regulation PlantMESH : Genes PlantMESH : Alkyl and Aryl TransferasesMESH : Stress Physiological030304 developmental biologyMESH : FMN ReductaseAlkyl and Aryl TransferasesArabidopsis ProteinsIron deficiencyNitric oxideNicotianaminebiology.organism_classificationMESH: Adaptation PhysiologicalMESH: DNA BacterialMESH : Gene ExpressionEnzymechemistryMESH: FMN ReductaseMESH: Plants Genetically ModifiedFerritinsMutationbiology.proteinMESH : FerritinsAgronomy and Crop ScienceMESH : Gene Expression Regulation Plant010606 plant biology & botany
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The FBN2 gene: new mutations, locus-specific database (Universal Mutation Database FBN2), and genotype-phenotype correlations.

2009

International audience; Congenital contractural arachnodactyly (CCA) is an extremely rare disease, due to mutations in the FBN2 gene encoding fibrillin-2. Another member of the fibrillin family, the FBN1 gene, is involved in a broad phenotypic continuum of connective-tissue disorders including Marfan syndrome. Identifying not only what is in common but also what differentiates these two proteins should enable us to better comprehend their respective functions and better understand the multitude of diseases in which these two genes are involved. In 1995 we created a locus-specific database (LSDB) for FBN1 mutations with the Universal Mutation Database (UMD) tool. To facilitate comparison of …

Fibrillin-2MESH : Polymorphism GeneticFibrillin-1DNA Mutational AnalysisMESH : Genotype[SDV.GEN] Life Sciences [q-bio]/Geneticscomputer.software_genreMESH: Genotype0302 clinical medicineGenotypeDatabases GeneticMissense mutationCongenital contractural arachnodactylyMESH: DNA Mutational AnalysisGenetics (clinical)MESH: Databases GeneticRegulation of gene expressionGenetics0303 health sciencesDatabaseMESH : Gene Expression RegulationMicrofilament ProteinsPhenotypeMESH: Gene Expression RegulationBeals-Hecht syndrome3. Good healthINCMESH : PhenotypePhenotypeMESH : MutationFibrillinmusculoskeletal diseasesMESH: MutationGenotypeMESH : Microfilament Proteinsdatabase OFFICIAL JOURNAL wwwhgvsorg & 2008 WILEY-LISSLocus (genetics)fibrillinMESH : DNA Mutational AnalysisBiologyFibrillinsMESH: PhenotypeMESH: Sequence Homology Nucleic Acidcongenital contractural arachnodactyly03 medical and health sciencesMESH: Microfilament ProteinsSequence Homology Nucleic AcidMESH: Polymorphism GeneticGeneticsmedicineHumansMESH : Sequence Homology Nucleic AcidFBN2CCAMESH : Databases GeneticGene030304 developmental biology[SDV.GEN]Life Sciences [q-bio]/GeneticsPolymorphism GeneticMESH: HumansMESH : Humansmedicine.diseaseGene Expression RegulationMutation[ SDV.GEN ] Life Sciences [q-bio]/Geneticscomputer030217 neurology & neurosurgery
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